Webb Therapy Uncategorized The Psychology of Gossiping – in a snapshot

The Psychology of Gossiping – in a snapshot

Gossiping is a universal social behaviour that involves the giving and receiving of information about others, generally perceived as having a negative effect on social groups and it is commonly sensationalistic in manner. The psychology of gossiping encompasses various aspects of human behaviour, including social interaction, communication, and interpersonal relationships.

Gossiping serves several psychological functions, such as forming and maintaining social bonds, establishing group norms, and conveying social information. Understanding the psychology of gossiping requires an examination of the underlying motivations, cognitive processes, and social dynamics involved in this behaviour.

One of the primary psychological functions of gossiping is its role in social bonding. According to evolutionary psychologists, gossiping may have evolved as a mechanism for monitoring and regulating social relationships within groups. By sharing information about others, individuals can establish and reinforce alliances, as well as identify potential threats or allies within their social networks. Gossiping also serves as a form of social currency, allowing individuals to exchange information and build rapport with others.

Furthermore, gossiping can be driven by intrinsic motivations related to curiosity and entertainment. People are naturally drawn to stories about others, particularly those involving conflict, romance, or scandal. This inclination toward sensationalistic narratives reflects the human tendency to seek novelty and emotional arousal through storytelling. From a psychological perspective, gossiping can be seen as a means of satisfying these innate cognitive and emotional needs.

In addition to its role in social bonding and entertainment, gossiping serves as a mechanism for transmitting social information and enforcing group norms. Through gossip, individuals communicate expectations and judgements regarding behaviour, values, and social roles within their communities. Gossip can function as a form of informal social control by publicly sanctioning or condemning certain behaviours, thereby influencing the conduct of group members.

The psychology of gossiping involves considerations of ethical and moral implications. While gossip can facilitate social cohesion and information sharing, it can also lead to negative consequences such as reputational damage, interpersonal conflict, disharmony, and breaches of privacy. Understanding the psychological mechanisms underlying gossiping can shed light on the ethical dilemmas associated with this behaviour and inform strategies for promoting responsible communication within social contexts.

Gossiping can indeed be malicious, as it involves spreading rumors or information about others that may be harmful, untrue, or damaging to their reputation. Malicious gossip can have serious consequences for the individuals involved, leading to damaged relationships, loss of trust, and even psychological harm. It is important to understand the impact of malicious gossip and the ethical considerations surrounding the spread of such information.

Malicious gossip is often driven by negative intentions, such as jealousy, resentment, or a desire to harm someone’s reputation. It can take various forms, including spreading false information about an individual’s personal life, career, or character. In some cases, malicious gossip may be used as a tool for bullying or manipulation, with the intent to undermine someone’s social standing or credibility.

The effects of malicious gossip can be far-reaching. It can lead to strained relationships, social ostracism, and damage to one’s professional reputation. In extreme cases, it can even result in legal action if the spread of false information causes tangible harm to an individual’s livelihood or well-being.

In summary, the psychology of gossiping encompasses various psychological functions, including its role in social bonding, entertainment, information transmission, and norm enforcement. By examining the underlying motivations, cognitive processes, and social dynamics involved in gossiping, researchers can gain insights into the complexities of human social behavior and interpersonal communication.

References:

Adler, R., & Proctor II, R. F. (2014). Looking out/looking in (14th ed.). Cengage Learning. (Print)

Dunbar, R.I.M. “Gossip in Evolutionary Perspective.” Review of General Psychology (Print)

Foster E.K., & Campbell W.K. “The Psychology of Gossip: A Review.” Social Psychological Review (Print)

Kniffin K.M., & Wilson D.S. “Evolutionary Perspectives on Gossip.” Social Psychology Quarterly (Print)

Kowalski, R. M., Limber, S. P., & Agatston, P. W. (2012). Cyberbullying: Bullying in the digital age (2nd ed.). Wiley-Blackwell. (Print)

Manning, J., & Levine, L. J. (2016). The psychology of social media: Why we like, share, comment and keep coming back. Routledge. (Print)

Robbins M.L., & Karan A. “Gossip: The Good, The Bad & The Ugly.” Journal of Applied Social Psychology (Print)

Salmivalli, C., & Graham-Kevan, N. (Eds.). (2019). Intimate partner violence: New perspectives in research and practice. Routledge. (Print)

Smith, P., & Steffgen, G. (Eds.). (2013). Cyberbullying through the new media: Findings from an international network. Psychology Press. (Print)

Sommerfeld R.D., & Jordan J.J. “The Evolutionary Foundations of Gossip.” Biological Theory (Print)

Related Post

There’s nothing ‘fake’ about ‘faking it until you make it’There’s nothing ‘fake’ about ‘faking it until you make it’

When to Fake It Till You Make It (and When You Shouldn’t)

Faking it for the right reasons can change you for the better. Here’s why.

Posted Jun 27, 2016By Amy Morin

One day, a client came to see me because she felt socially awkward. She knew that her inability to make small talk was holding her back both personally and professionally. As a shy person, she hated going to networking events. But making connections was vital to her career.I asked, “What do you usually do when you go to a networking event?” She said, “I stand awkwardly off to the side and wait to see if anyone will come talk to me.” I asked her, “What would you do differently if you felt confident?” and she said, “I’d initiate conversation and introduce myself to people.”

Right then and there, she discovered the solution to her problem: If she wanted to feel more confident, she had to act more confident. That wasn’t quite what she wanted to hear. She’d hoped for a solution that would immediately make her feel more confident. But the key to becoming more comfortable in social situations is practice.Her instinct was to wait until she felt more confident, but that confidence wasn’t going to magically appear out of thin air—especially if she was standing around by herself. However, if she started talking to people like a confident person, she’d have an opportunity to experience successful social interactions, and each of these would boost her confidence.

Acting “As If”

Acting “as if” is a common prescription in psychotherapy. It’s based on the idea that if you behave like the person you want to become, you’ll become like this in reality:

1. If you want to feel happier, do what happy people do—smile.

2. If you want to get more work done, act as if you are a productive person.

3. If you want to have more friends, behave like a friendly person.

4. If you want to improve your relationship, practice being a good partner.Too often we hesitate to spring into action. Instead, we wait until everything feels just right or until we think we’re ready. But research shows that changing your behavior first can change the way you think and feel.

The Biggest Mistake Most People Make

Faking it until you make it only works when you correctly identify something within yourself that’s holding you back. Behaving like the person you want to become is about changing the way you feel and the way you think.If your motives are to prove your worth to other people, however, your efforts won’t be successful, and research shows that this approach actually backfires. A study published in the Journal of Consumer Research found that people who tried to prove their worth to others were more likely to dwell on their shortcomings. Ambitious professionals who wore luxury clothing in an effort to appear successful, and MBA students who wore Rolex watches to increase their self-worth just ended up feeling like bigger failures. Even worse, their attempts to project an image of success impaired their self-control. They struggled to resist temptation when they tried to prove that they were successful. Putting so much effort into faking it used up their mental resources and interfered with their ability to make good choices.

How to “Fake It” the Right Way

Acting “as if” doesn’t mean being phony or inauthentic. It’s about changing your behavior first and trusting the feelings will follow. As long as your motivation is in the right place, faking it until you make it can effectively make your goals become reality. Just make sure you’re interested in changing yourself on the inside, not simply trying to change other people’s perceptions of you.

Understanding Addiction: A Modern, Integrative PerspectiveUnderstanding Addiction: A Modern, Integrative Perspective

Abstract

Addiction is a complex, multifaceted phenomenon that has been described variously as a disease, disorder, syndrome, obsessive-compulsive behaviour, learned behaviour, or spiritual malady. Modern scientific understanding emphasises addiction as a chronic brain disorder shaped by neurobiological changes, learning, and social context. This article examines each conceptualisation and presents an integrated definition that aligns with current neuroscience, psychological, and public health evidence.

Conceptualising Addiction: Labels and Their Accuracy

No single label fully captures addiction’s complexity; each highlights certain truths while overlooking others.

Disease

From a medical perspective, disease is the closest match. Addiction involves persistent neurobiological changes in reward, stress, and self-control circuits, increases relapse risk over years, and shows substantial genetic vulnerability (~50–60%) (NIDA, 2018; Heilig et al., 2021). Treatments improve outcomes but rarely “cure” the condition. This framing is used by the American Society of Addiction Medicine (ASAM), NIDA, WHO ICD-11, and DSM-5-TR (as “Substance Use Disorder”) (NIDA, 2018).

Disorder

Disorder is also scientifically accurate and slightly less medicalised. DSM-5’s “Substance Use Disorder” captures behavioural, psychological, and biological criteria and recognises functioning and harm rather than framing addiction strictly as a lifelong disease (Heather, n.d.; Heilig et al., 2021).

Syndrome

Addiction may be described as a syndrome because it is a cluster of symptoms with behavioural and physiological manifestations, without a single causative factor. However, the term is too generic for practical use outside clinical texts (Blithikioti et al., 2025).

Obsessive and Compulsive Learned Behaviour

Addiction involves learning, habit formation, and compulsion through reinforcement of rewarding behaviours (Hyman, 2005; Hausotter, 2013). Yet describing it solely as learned behaviour ignores genetic predisposition, neuroadaptation, withdrawal, and social factors.

Spiritual Malady

Some mutual-aid traditions characterise addiction as a spiritual malady. While this may be meaningful for individuals, it is not scientifically explanatory: addiction can be adequately explained via biological, psychological, and social mechanisms (Lewis, 2017).

Modern Integrative Definition

The most accurate contemporary description of addiction is:
“A chronic, relapsing disorder of brain circuits involved in reward, stress, and self-control, shaped by learning, environment, and social context”.

This definition encompasses:

  • Disease/disorder: medical accuracy
  • Learned behaviour and compulsion: neuroscience and behavioural accuracy
  • Social determinants: public health relevance
  • Flexibility for personal or spiritual interpretations

In short, addiction is best understood as a bio-psycho-social condition that is treatable and sometimes reversible, rather than a deterministic, lifelong curse.

Neurobiology: Why Addiction Is Considered a Brain Disorder

Repeated substance use alters structural and functional brain circuits involved in reward, stress, motivation, memory, and self-control (Nwonu et al., 2022; NIDA, 2018). These changes can persist long after use stops, explaining why addiction is more than a matter of “bad habits” or weak will (NIDA, 2025).

Chronicity and Relapse

Addiction is often chronic and relapsing. Even after long periods of abstinence, cues and stressors can trigger relapse (Meurk et al., 2014; SAMHSA, 2023). Key regions implicated include the basal ganglia (habit formation), extended amygdala (stress), and prefrontal cortex (decision-making) (Kirby et al., 2024). Nevertheless, many individuals achieve stable remission, highlighting heterogeneity in clinical outcomes (Heilig et al., 2021).

Learning, Memory, and Habit Formation

Addiction exploits neural mechanisms of learning and memory: rewarding behaviours are repeated and consolidated into habits, with cues triggering compulsive responses even when the substance’s reward diminishes (Hausotter, 2013; Lewis, 2017). This intertwines biological disorder and learned behaviour.

Critiques and Limitations

Some scientists caution that framing addiction strictly as a brain disease is simplistic:

  • Brain changes may resemble those from other motivated behaviours (Lewis, 2017).
  • Many recover without formal treatment (Heilig et al., 2021).
  • Social, environmental, and psychological factors are crucial to understanding addiction (Blithikioti et al., 2025).

Thus, while the disease model is powerful, it does not fully represent addiction’s heterogeneity or socio-psychological dimensions.

Implications for Treatment

Addiction is treatable, not simply curable. Interventions combining pharmacological and behavioural approaches, alongside social support, can foster long-term recovery (Liu & Li, 2018; Heilig et al., 2021). Like other chronic conditions, management — rather than elimination — is often the realistic goal (NIDA, 2018). Neural circuits can gradually readjust, particularly when environmental and personal factors support recovery.

Conclusion

Addiction is a learned, compulsive brain disorder with chronic potential, shaped by neurobiological, psychological, social, and environmental factors. Recognising addiction as both a disorder and a behavioural learning condition avoids extremes: it is neither an unchangeable fate nor merely a moral failing. This integrated perspective supports nuanced understanding, compassionate care, and effective treatment strategies.


References

Blithikioti, C., Fried, E. I., Albanese, E., Field, M., & Cristea, I. A. (2025). Reevaluating the brain disease model of addiction. The Lancet Psychiatry, 12(6), 469–474. https://doi.org/10.1016/S2215-0366(25)00060-4

Hausotter, W. (2013). Neuroscience and understanding addiction. Addiction Technology Transfer Center (ATTC) Network. https://attcnetwork.org/neuroscience-and-understanding-addiction

Heather, N. (n.d.). What’s wrong with the brain disease model of addiction (BDMA)? Addiction Theory Network. https://addictiontheorynetwork.org/brain-disease-model-of-addiction

Heilig, M., MacKillop, J., Martinez, D., Rehm, J., Leggio, L., & Vanderschuren, L. J. M. J. (2021). Addiction as a brain disease revised: Why it still matters, and the need for consilience. Neuropsychopharmacology, 46(10), 1715–1723. https://doi.org/10.1038/s41386-020-00950-y

Hyman, S. E. (2005). Addiction: A disease of learning and memory. The American Journal of Psychiatry, 162(8), 1414–1422. https://doi.org/10.1176/appi.ajp.162.8.1414

Kirby, E. D., Glenn, M. J., Sandstrom, N. J., & Williams, C. L. (2024). Neurobiology of addiction (Section 14.5). In Introduction to Behavioral Neuroscience. OpenStax. https://socialsci.libretexts.org/…/14.05:_Neurobiology_of_Addiction

Leshner, A. I. (1997). Addiction is a brain disease, and it matters. Science, 278(5335), 45–47. https://doi.org/10.1126/science.278.5335.45

Lewis, M. (2017). Addiction and the brain: Development, not disease. Neuroethics, 10(1), 7–18. https://doi.org/10.1007/s12152-016-9293-4

Liu, J. F., & Li, J. X. (2018). Drug addiction: A curable mental disorder? Acta Pharmacologica Sinica, 39(12), 1823–1829. https://doi.org/10.1038/s41401-018-0180-x

Meurk, C., Carter, A., Partridge, B., Lucke, J., & Hall, W. (2014). How is acceptance of the brain disease model of addiction related to Australians’ attitudes towards addicted individuals and treatments for addiction? BMC Psychiatry, 14, 373. https://doi.org/10.1186/s12888-014-0373-x

National Institute on Drug Abuse. (2018). Drugs, brains, and behavior: The science of addiction (Rev. ed.). https://irp.nida.nih.gov/…/NIDA_DrugsBrainsAddiction

Nwonu, C. N. S., Nwonu, P. C., & Ude, R. A. (2022). Neurobiological underpinnings in drug addiction. West African Journal of Medicine, 39(6), 874–884. https://pubmed.ncbi.nlm.nih.gov/36063103

Substance Abuse and Mental Health Services Administration. (2023). What is substance use disorder? U.S. Department of Health and Human Services. https://www.samhsa.gov/substance-use/what-is-sud

How often do you feel lonely?How often do you feel lonely?

Dr Rangan Chatterjee chats with Dr Gabor Maté about his radical findings based on decades of work with patients. We’re currently living in a culture that doesn’t meet our human needs. Maté and Chatterjee delve into how our emotional stress can translate into physical chronic illnesses, and how loneliness and a lack of meaningful connection are on the rise, as are the rates of autoimmune disease and addiction.